Review highlights the growing evidence that aging is linked to reduced expression of long genes, offering insights into potential anti-aging therapies based on gene length.
By Dr. Chinta SidharthanReviewed by Susha Cheriyedath, M.Sc.Sep 19 2024 Review explores the connection between gene length and aging, summarizing recent findings that link reduced long- gene expression to age-related decline and potential anti-aging strategies.
Long-gene expression Understanding the genetic underpinnings of aging has long been one of the major focal points of biological science research. Numerous studies aim to identify the genes that play a central role in aging. However, identifying the genetic basis of aging has been a challenge. Insights on aging from revisited data Early attempts by stem cell biologist Ander Izeta from the Biogipuzkoa Research Institute in Spain failed to uncover any gene expression patterns in aging. However, his research found a new lease of life when he encountered data from a 2016 study by a molecular geneticist called Jan Hoeijmakers from Erasmus University in the Netherlands.
Related StoriesLater, Stoeger and colleagues reported that the use of anti-aging treatments such as resveratrol, senolytics, and rapamycin increased the expression of long genes in aging mice. This finding further confirmed the malleable nature of long-gene expression, suggesting that anti-aging therapies could potentially reverse age-related transcriptional decline.
Genes Genetic Cockayne Syndrome DNA Gene Gene Expression Genome Research RNA Syndrome Transcription Xeroderma Pigmentosum
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