Researchers at Rutgers and Emory University are gaining insights into how schizophrenia develops by studying the strongest-known genetic risk factor.
. This dysfunction can cause energy shortfalls in the brain and result in psychiatric symptoms and disorders.
."The interplay between mitochondrial dynamics and neuronal maturation is an important area for additional detailed and rigorous study." "For genetic variants associated with schizophrenia, we want to understand the primary pathology at the," said Ryan Purcell, assistant professor of cell biology at Emory University School of Medicine and co-lead author of the study."This gives us a foothold, which may help cut through schizophrenia's polygenic complexity and better understand the neurobiology."
The finding that various schizophrenia-associated chromosomal deletions impair mitochondria runs counter to an expectation in the field that such mutations should alter proteins in the synapses that connect neurons. However, mitochondria are critical for energy-hungry synapses' function—so these models may not be in conflict.
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