In 1937, President Franklin Roosevelt signed the National Cancer Act, launching a nationwide effort to combat the disease.
University of Alabama at BirminghamNov 7 2024 Eighty-seven years later, despite significant progress, cancer treatment often falls short, with 50 to 80 percent of patients not responding to treatment and more than 600,000 cancer deaths annually in the United States.
To tackle this complexity, a research team at the University of Alabama at Birmingham led by Anindya Dutta, Ph.D., professor and chair of the UAB Department of Genetics, sought to identify patterns within this apparent randomness.
The researchers applied UAB36 to predict the expression of genes linked with the resistance of breast cancer against tamoxifen, a drug widely used for breast cancer treatment. UAB36 consistently showed a higher efficacy compared to a single gene approach. UAB36 also outperformed established gene signatures like ENDORSE and PAM50 in its correlation with tamoxifen resistance in breast cancer cells.
Gene Breast Cancer Cancer Treatment Cell Drugs Efficacy Gene Expression Genes Genetic Genetics Genomics Immune Response Medicine Oncology Research Tamoxifen
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