Science, Space and Technology News 2024
A new study has pinpointed brain neurons that separate the sensation of fullness from nausea in obesity drugs, paving the way for treatments that suppress appetite without causing adverse effects.
The next chapter in the story of headline-making popular obesity drugs could focus on understanding the physical sensation of fullness after eating compared to the brain’s regulation of nausea. Researchers at thehave identified a group of neurons in the brain that manage food consumption without inducing nausea in animal models, distinguishing the beneficial aspects of these drugs from their side effects., describes two distinct neural circuits that govern different effects of the same drug.
Activating GLP1 receptor-expressing neurons in the nucleus tractus solitarius increase sateity without causing nausea or aversion. Credit: Alisha Acosta, Monell Chemical Senses Center“One of the barriers to drug treatments for obesity is side effects such as nausea and vomiting,” said senior author Amber L. Alhadeff, PhD, Monell Assistant Member. “We did not have a good idea of whether these unpleasant side effects are related or necessary for the weight-loss effects.
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