A new study published in JACC reveals that even brief exposure to elevated ozone levels can reduce blood oxygen saturation, trigger hypoxia-related biomarkers, and increase arterial stiffness, highlighting the link between ozone pollution and cardiovascular health.
A new study published in the Journal of the American College of Cardiology (JACC) reveals a potential link between ozone (O3) exposure and arterial stiffness. Researchers discovered that even brief periods of elevated ozone levels can lead to decreased blood oxygen saturation, triggering biomarkers associated with hypoxia (oxygen deficiency) and increasing arterial stiffness.
This study provides compelling evidence of a novel connection between ozone exposure and cardiovascular health, shedding light on the independent role of ozone in causing damage to the heart and blood vessels. The research was conducted in a high-altitude setting, allowing scientists to isolate the effects of ozone from other common air pollutants. This unique approach provides valuable insights into the specific impact of ozone on the cardiovascular system. Dr. Harlan Krumholz, MD, SM, Editor-in-Chief of JACC, emphasizes the growing global concern surrounding ozone pollution and its potential to contribute to a range of cardiovascular diseases, including ischemic heart disease, stroke, heart failure, and atherosclerosis. Hypoxia, or a lack of oxygen in the body, is recognized as a key factor in the development of cardiovascular problems linked to ozone exposure. This study's findings underscore the importance of understanding and mitigating the risks associated with ozone pollution to protect public health
Ozone Pollution Cardiovascular Disease Arterial Stiffening Hypoxia Environmental Health
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