Urgent warning to anyone who’s had Covid – bug ‘increases risk of silent killers by 55%’
Patients’ hearts had higher levels of inflammation and unstable levels of calcium, which is needed to allow the muscle to contract.They also looked at mice infected with Covid to see how it changed compared to those without the virus.a high temperature or shivering – a high temperature means you feel hot to touch on your chest or back
a new, continuous cough – this means coughing a lot for more than an hour, or 3 or more coughing episodes in 24 hours
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The brain reacting to COVID-19: analysis of the cerebrospinal fluid proteome, RNA and inflammation - Journal of NeuroinflammationPatients with COVID-19 can have a variety of neurological symptoms, but the active involvement of central nervous system (CNS) in COVID-19 remains unclear. While routine cerebrospinal fluid (CSF) analyses in patients with neurological manifestations of COVID-19 generally show no or only mild inflammation, more detailed data on inflammatory mediators in the CSF of patients with COVID-19 are scarce. We studied the inflammatory response in paired CSF and serum samples of patients with COVID-19 (n = 38). Patients with herpes simplex virus encephalitis (HSVE, n = 10) and patients with non-inflammatory, non-neurodegenerative neurological diseases (n = 28) served as controls. We used proteomics, enzyme-linked immunoassays, and semiquantitative cytokine arrays to characterize inflammatory proteins. Autoantibody screening was performed with cell-based assays and native tissue staining. RNA sequencing of long-non-coding RNA and circular RNA was done to study the transcriptome. Proteomics on single protein level and subsequent pathway analysis showed similar yet strongly attenuated inflammatory changes in the CSF of COVID-19 patients compared to HSVE patients with, e.g., downregulation of the apolipoproteins and extracellular matrix proteins. Protein upregulation of the complement system, the serpin proteins pathways, and other proteins including glycoproteins alpha-2 and alpha-1 acid. Importantly, calculation of interleukin-6, interleukin-16, and CXCL10 CSF/serum indices suggest that these inflammatory mediators reach the CSF from the systemic circulation, rather than being produced within the CNS. Antibody screening revealed no pathological levels of known neuronal autoantibodies. When stratifying COVID-19 patients into those with and without bacterial superinfection as indicated by elevated procalcitonin levels, inflammatory markers were significantly (p | 0.01) higher in those with bacterial superinfection. RNA sequencing in the CSF revealed 101 linear RNAs comprising mess
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COVID-19: Interactive risk communication formats can help overcome vaccine hesitancy, finds studyInteractive risk communication formats can be more effective than conventional text-based formats in overcoming vaccine hesitancy and building public trust. These are the findings of a study conducted by researchers at Charité-Universitätsmedizin Berlin and the Max Planck Institute for Human Development in the context of COVID-19 vaccination. The study's findings have been published in JAMA Network Open.
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Mass. Reports 4,470 New COVID-19 Cases, 102 Deaths This WeekMassachusetts health officials reported 4,470 new COVID-19 cases and 102 new deaths in the last week, with the new data released Thursday. In total, there have been 2,014,494 cases and 22,132 deaths since the start of the coronavirus pandemic.
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Past COVID infection 'as good as vaccines' at preventing severe illnessWhile the research suggests that natural immunity could be just as good as vaccines at preventing serious illness from COVID-19 infection, the study's authors encourage people to still get vaccinated, to avoid any complications from any initial infections.
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Research looks to determine prevalence of organ impairment in long-COVID patientsResearch looks to determine prevalence of organ impairment in long-COVID patients Research Coronavirus Disease COVID19 LongCOVID PASC editorjrsm LivUni UniOfHull UniofOxford ucl
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